Omics analysis of mouse brain models of human diseases

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• 作者：Vé ; ronique Paban^a ; ^{veronique.paban@univ-amu.fr} ; Bé ; atrice Loriod^b ; Claude Villard^c ; Luc Buee^d ; David Blum^d ; Susanna Pietropaolo^e ; ^f ; Yoon H. Cho^e ; ^f ; Sylvie Gory-Faure^g ; ^h ; ⁱ ; Elodie Mansour^a ; Ali Gharbi^a ; Bé ; atrice Alescio-Lautier^a
• 关键词：Adam23 ; ADAM metallopeptidase domain 23 ; Alcam ; activated leukocyte cell adhesion molecule ; Apoe ; apolipoprotein E ; Atp5l ; ATP synthase ; H  ; + transporting ; mitochondrial Fo complex subunit G ; Cct8 ; chaperonin containing TCP1 subunit 8 ; Cd82 ; CD82 molecule ; Ckb ; creatine kinase B ; Cntn1 ; contactin 1 ; Cox7b ; cytochrome C oxidase subunit 7B ; Cst7 ; cystatin F ; Galp ; galanin like peptide ; Gfap ; glial fibrillary acidic protein ; Gnb1 ; G protein subunit beta 1 ; Gps1 ; G protein pathway suppressor 1
• 刊名：Gene
• 出版年：2017
• 出版时间：5 February 2017
• 年：2017
• 卷：600
• 期：Complete
• 页码：90-100
• 全文大小：664 K
• 卷排序：600

文摘

A common molecular signature does not exist across brain diseases. Only one major common function exists across brain diseases. Hubs/bottlenecks represented < 20% of all disease genes/proteins.